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Cell growth was inhibited regardless of p53 functionality.
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The decrease in cell viability and increase in population were both found to be dose-dependent, but an 80mm metformin dose caused apoptosis in both cell lines.
“diabetic patients are associated with a higher hcc risk, which is attenuated by the use of metformin in a dose-dependent and duration-dependent manner,” the researchers wrote.
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“… to the best of our knowledge, this is the first time that metformin has been demonstrated to inhibit cell growth through cell cycle g0/g1 arrest in hepatoma cell lines … metformin, generally considered a safe, well-tolerated and relatively inexpensive drug for type 2 diabetes, may represent a new strategy to improve treatment of hepatoma.
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